soluble human dll4 (R&D Systems)
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R&D Systems
soluble human dll4
Soluble Human Dll4, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 60 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/recombinant+soluble+human+dll4/Recombinant+Human+DLL4+His-tag+Protein/pm20182391-32-29-32
Average 94 stars, based on 60 article reviews
Soluble Human Dll4, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 60 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/recombinant+soluble+human+dll4/Recombinant+Human+DLL4+His-tag+Protein/pm20182391-32-29-32
Average 94 stars, based on 60 article reviews
soluble human dll4 - by Bioz Stars,
2026-09
94/100 stars
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Related Articles
Generated:Article Title: Blockade of Delta-Like Ligand 4 Signaling Inhibits Both Growth and Angiogenesis of Pancreatic Cancer Article Snippet: Objectives: The Notch signaling pathway is evolutionarily conserved and regulates cell-fate decisions in a variety of organ development.. Previous studies have shown that delta-like ligand 4 (DLL4), one of transmembranous Notch ligands, is up-regulated at the site of tumor growth, especially of tumor angiogenesis.. In this study, we examined the effects of the DLL4-Notch signaling on tumor angiogenesis using the neutralizing monoclonal antibodies against DLL4. Recombinant:Article Title: Blockade of Delta-Like Ligand 4 Signaling Inhibits Both Growth and Angiogenesis of Pancreatic Cancer Article Snippet: Objectives: The Notch signaling pathway is evolutionarily conserved and regulates cell-fate decisions in a variety of organ development.. Previous studies have shown that delta-like ligand 4 (DLL4), one of transmembranous Notch ligands, is up-regulated at the site of tumor growth, especially of tumor angiogenesis.. In this study, we examined the effects of the DLL4-Notch signaling on tumor angiogenesis using the neutralizing monoclonal antibodies against DLL4. |